Whether it is harmful mold on the walls?

Many years ago, during the war years, mold has saved mankind, giving him the first antibiotic - penicillin. Today consider that the mold in damp buildings is a factor contributing to respiratory morbidity. Finnish scientists have answered the question: how and to what extent, affects of mold in buildings on respiratory morbidity from the point of view of evidence-based medicine.

A meta-analysis published in the "Cochrane Collaboration", covers 51 a study on the impact of the fungus in damp areas on respiratory morbidity in children and adults.

The conclusions that scientists have made amazing. Today there is no scientific data on the relationship of contact with mold on the walls and broncho-pulmonary disease. Previous studies that argued about the increasing incidence of contact with mold may not be evidence-based, as it does not have a proper control group and do not take into account many other factors. For example, mold in cheese production facility, with staff in addition to the fungus in contact with the technical dust, or not considered a factor in the area, which in itself can significantly affect the incidence.

Based on data obtained from previously conducted studies that take into account other harmful factors and have a control comparison group, the researchers conclude: mould on the walls has no effect on broncho-pulmonary morbidity in adults and in children, elimination of contact with the mold reduces health care seeking about respiratory diseases.

The researchers did not assess the impact of mold on the development of asthma, as this disease can develop for decades, and today is available the analysis is less long-term observations.

Source: Sauni R, Uitti J, Jauhiainen M, Kreiss To Sigsgaard T, Verbeek JH. Remediating buildings damaged damp and mould to prevent or reduce symptoms of respiratory diseases respiratory tract infections and asthma. Cochrane database of systematic reviews 2011, issue 9. Article No.: CD007897.DOI: 10.1002/14651858.CD007897.pub2.

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