Immune cells are not only the system protect the body from harmful microbes. Together with other cells, including our native bacteria symbionts, they participate in the regulation of normal functioning of our body.
Based on the results of new research, published recently in Nature Medicine, a kind of "negotiation" between immune B-cells, intestinal cells and living in it the germs, all combine to assist the body in regulating the fat intake. New discoveries will probably explain why the people who get AIDS, as well as suffering from other chronic infectious diseases, cease to eat normally, and how this factor can be corrected.
Earlier it was observed that mice, which suffer from a lack of immune B-cells, much worse metabolize fats than their healthy relatives. And recently, our compatriots Andrew Morgana and Natalia A. Sulzenko, currently working in the United States showed that it is responsible protein immunoglobulin IgA.
As it turned out, the group of intestinal cells (epithelial cells) make a choice between two modes of operation: or metabolism - extraction of energy by the breakdown of food, or self-protection. Yet inexplicably, IgA proteins is favored by the fact that the symbionts-bacteria "send" signals in the cells and set the correct "routine" of their work.
Immune-biologist Andrew Gewirtz (USA), performs a comparison between the similar role of cells of the immune system and the police: "the Police, the work included not only when committed crimes". Also immune cells continuously maintain health and good condition of the body.